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The Skinny on Fats, Great Read


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https://www.westonaprice.org/health-topics/know-your-fats/the-skinny-on-fats/#intro

 

Dietary SFA consumption is generally not associated with increased CVD incidence or mortality.

 

"Conclusions: Saturated fats are not associated with all cause mortality, CVD, CHD, ischemic stroke, or type 2 diabetes, but the evidence is heterogeneous with methodological limitations"

 

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4532752/

 

"Conclusions: A meta-analysis of prospective epidemiologic studies showed that there is no significant evidence for concluding that dietary saturated fat is associated with an increased risk of CHD or CVD."

 

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2824152/

 

The massive PURE study made similar findings in 2017

 

"INTERPRETATION: High carbohydrate intake was associated with higher risk of total mortality, whereas total fat and individual types of fat were related to lower total mortality. Total fat and types of fat were not associated with cardiovascular disease, myocardial infarction, or cardiovascular disease mortality, whereas saturated fat had an inverse association with stroke."

 

https://www.ncbi.nlm.nih.gov/pubmed/28864332

 

 

Replacing dietary SFA with PUFA, specifcially n-6 PUFA, does not appear (at least consistently) to reduce CVD events, mortality or overall mortality

 

"CONCLUSION: Available evidence from adequately controlled randomised controlled trials suggest replacing SFA with mostly n-6 PUFA is unlikely to reduce CHD events, CHD mortality or total mortality. The suggestion of benefits reported in earlier meta-analyses is due to the inclusion of inadequately controlled trials. These findings have implications for current dietary recommendations."

 

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5437600/

 

 

And Uri Ravnskov has been questioning the diet-heart hypothesis for a long time

 

"Among 21 cohort studies of CHD including 28 cohorts, CHD patients had eaten significantly more SFA in three cohorts and significantly less in one cohort than had CHD-free individuals; in 22 cohorts no significant difference was noted. In three cohorts, CHD patients had eaten significantly more PUFA, in 24 cohorts no significant difference was noted. In three of four cohort studies of atherosclerosis, the vascular changes were unassociated with SFA or PUFA; in one study they were inversely related to TF. No significant differences in fat intake were noted in six case-control studies of CVD patients and CVD-free controls; and neither total or CHD mortality were lowered in a meta-analysis of nine controlled, randomized dietary trials with substantial reductions of dietary fats, in six trials combined with addition of PUFA. The harmful effect of dietary SFA and the protective effect of dietary PUFA on atherosclerosis and CVD are questioned."

 

https://www.ncbi.nlm.nih.gov/pubmed/9635993

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